What a qEEG can reveal about mood and emotional regulation.
Mood is not a single thing in the brain. It's the downstream signature of how the prefrontal cortex, the limbic system, and the brain's arousal networks are talking to each other.
Frontal alpha asymmetry
One of the most-studied EEG markers in mood research is the balance of alpha activity between the left and right frontal cortices. People with depression often show greater right-frontal activity (less left-frontal alpha suppression), a pattern associated with avoidance and reduced approach motivation. Research dating back to Davidson and colleagues in the 1990s has reproduced this finding in multiple populations [1, 2].
Beta excess and over-arousal
Many people who present with anxiety show elevated high-beta activity across central and frontal sites — a marker of cortical over-arousal that mirrors the felt experience of being "wired and tired." This pattern often sits alongside elevated baseline heart rate and reduced heart-rate variability [3].
Network connectivity
More recent research has moved beyond single-channel findings and into how different brain regions are working together. In trauma-related conditions, we frequently see altered connectivity between the default-mode network (involved in self-referential thinking) and the salience network (involved in threat detection). This is why people with PTSD often describe being unable to stop scanning, even when nothing is actually wrong [4].
What this means for a clinical plan
None of these markers diagnose anything on their own. But put together with the conversation, the history, and what your other clinicians have observed, they help us build a personalised neurofeedback and biofeedback plan that targets the specific patterns showing up — rather than running everyone through the same protocol.